CV Michael Hensel
Michael Hensel studied Biology at the University of Osnabrück and graduated with a PhD in Microbiology in 1993. He did his postdoctorate at the Hammersmith Hospital in London and returned to Germany in 1996 to work as a group leader at the Max von Pettenkofer-Institute in München. In 2000, he habilitated in Microbiology and accepted a professorship position in Microbiology at the FAU Erlangen-Nürnberg. In 2009, he took over the chair of Microbiology at the University of Osnabrück.
- Manipulation of Host Cell Organelles by Intracellular Pathogens. Int J Mol Sci 22:6484.
- Single cell analyses reveal distinct adaptation of typhoidal and non-typhoidal Salmonella enterica serovars to intracellular lifestyle. PLoS Pathog 17:e100931 9.
- A trafficome-wide RNAi screen reveals deployment of early and late secretory host proteins and the entire late endo-/lysosomal vesicle fusion machinery by intracellular Salmonella. PLoS Pathog 16:e1008220.
- Role of the ESCRT-III complex in controlling integrity of the Salmonella-containing vacuole. Cell Microbiol 22:e13176.
- Presence of SopE and mode of infection result in increased Salmonella-containing vacuole damage and cytosolic release during host cell infection by Salmonella enterica. Cell Microbiol 22:e13155.
- Blocks in Tricarboxylic Acid Cycle of Salmonella enterica Cause Global Perturbation of Carbon Storage, Motility, and Host-Pathogen Interaction. mSphere 4:e00796-19.
- Self-Labeling Enzyme Tags for Analyses of Translocation of Type III Secretion System Effector Proteins. mBio 10:e00769-19.
- Structural and functional dissection reveals distinct roles of Ca2+-binding sites in the giant adhesin SiiE of Salmonella enterica. PLoS Pathog 13:e1006418. (2017)
- Reorganization of the endosomal system in Salmonella-infected cells: the ultrastructure of Salmonella-induced tubular compartments. PLoS Pathog 10:e1004374.
- Salmonella enterica invasion of polarized epithelial cells is a highly cooperative effort. Infect Immun 82:2657-2667